Abstract
The development of our current understanding of the molecular basis of susceptibility to rheumatoid arthritis is discussed with emphasis on newer information that expands the concept of the nature and role of a 'shared epitope.'
| Original language | English |
|---|---|
| Pages (from-to) | 761-783 |
| Number of pages | 23 |
| Journal | Rheumatic Disease Clinics of North America |
| Volume | 18 |
| Issue number | 4 |
| State | Published - 1992 |
| Externally published | Yes |