Targeting the NF-kB signaling pathway in chronic tendon disease

Medicine and Dentistry

• Disease 100%
• Surgery 100%
• Signal Transduction 100%
• Tendinosis 100%
• Tendon Disease 100%
• Fibroblast 100%
• Drive 50%
• Gene Expression Profiling 50%
• Cytokine 50%
• Immunocompetent Cell 50%
• Rotator Cuff 50%
• Rotator Cuff Injury 50%
• Stromal Cell 50%
• Gene Deletion 50%
• Musculoskeletal Disease 50%
• Humeral Head 50%
• Protein Serine Kinase 50%
• Nuclear Factor Kappa B 50%

Keyphrases

• NF-κB Signaling Pathway 100%
• IκB Kinase 100%
• Chronic Tendon Disease 100%
• NF-κB Pathway 60%
• Inflammation 40%
• Overexpression 40%
• Surgical Repair 40%
• Tendon Degeneration 40%
• Tendinopathy 40%
• Rotator Cuff Tendinopathy 40%
• Tendon Disorders 40%
• Tendon Fibroblast 40%
• Improved Outcomes 20%
• Clinically Significant 20%
• Inflammatory Cytokines 20%
• Healing 20%
• Nuclear factor-κB 20%
• Tears 20%
• Clinical Evidence 20%
• Medical Attention 20%
• Transcriptional Profiling 20%
• Innate Immune Cells 20%
• Tendon 20%
• Therapeutic Approaches 20%
• Tendon Healing 20%
• Genomic Deletion 20%
• Musculoskeletal Complaints 20%
• Stromal Cells 20%
• Conditional Knockout 20%
• Humeral Head 20%
• Serine Kinase 20%
• Therapeutic Candidates 20%
• NF-κB Signaling 20%
• Return Function 20%
• Human Tendon 20%
• Free Return 20%

Immunology and Microbiology

• Signal Transduction 100%
• Tendinitis 100%
• Tendinosis 100%
• Rotator Cuff 66%
• Fibroblast 66%
• Immunocompetent Cell 33%
• Proinflammatory Cytokine 33%
• Gene Deletion 33%
• Nuclear Factor Kappa B 33%
• Humeral Head 33%
• Gene Expression Assay 33%