Abstract
Spasmodic dysphonia (SD) is a focal laryngeal dystonia characterized by involuntary, action-induced spasms of the muscles controlling vocal fold motion. The laryngeal adductor muscles (lateral cricoarytenoid [LCA], interarytenoid, and possibly the cricothyroid and thyroarytenoid [TA]), abductor muscle (posterior cricoarytenoid [PCA]), or rarely both groups of muscles may be affected. Adductor SD is characterized by a harsh, strangled, or effortful voice (glottal fry) with irregular phonatory breaks secondary to vocal fold hyperadduction or spasm. The supraglottic structures may be hyperfunctional as well. Abductor SD presents as a breathy, effortful, hypophonic voice with irregular breaks following consonant voicing secondary to vocal fold hyperabduction. Although the exact etiology of SD is unclear, SD is now recognized as a neurological disorder of central processing.
| Original language | English |
|---|---|
| Title of host publication | Therapeutic Uses of Botulinum Toxin |
| Publisher | Humana Press |
| Pages | 109-121 |
| Number of pages | 13 |
| ISBN (Print) | 9781588299147 |
| DOIs | |
| State | Published - 2007 |
| Externally published | Yes |
Fingerprint
Dive into the research topics of 'Spasmodic dysphonia'. Together they form a unique fingerprint.Cite this
- APA
- Author
- BIBTEX
- Harvard
- Standard
- RIS
- Vancouver