Silence of the ROS

Judith Agudo; Brian D. Brown

doi:10.1016/j.immuni.2016.02.027

Silence of the ROS

Judith Agudo, Brian D. Brown

Research output: Contribution to journal › Short survey › peer-review

6 Scopus citations

Abstract

Reactive oxygen species (ROS) are generated during T cell activation and serve a signaling function but can also be damaging. In this issue of Immunity, Zhang et al. (2016) show that miR-23a prevents ROS-elicited necrosis by suppressing cyclophilin D (PPIF), a regulator of ROS escape from mitochondria.

Original language	English
Pages (from-to)	520-522
Number of pages	3
Journal	Immunity
Volume	44
Issue number	3
DOIs	https://doi.org/10.1016/j.immuni.2016.02.027
State	Published - 15 Mar 2016

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title = "Silence of the ROS",

abstract = "Reactive oxygen species (ROS) are generated during T cell activation and serve a signaling function but can also be damaging. In this issue of Immunity, Zhang et al. (2016) show that miR-23a prevents ROS-elicited necrosis by suppressing cyclophilin D (PPIF), a regulator of ROS escape from mitochondria.",

author = "Judith Agudo and Brown, {Brian D.}",

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year = "2016",

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T1 - Silence of the ROS

AU - Agudo, Judith

AU - Brown, Brian D.

PY - 2016/3/15

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N2 - Reactive oxygen species (ROS) are generated during T cell activation and serve a signaling function but can also be damaging. In this issue of Immunity, Zhang et al. (2016) show that miR-23a prevents ROS-elicited necrosis by suppressing cyclophilin D (PPIF), a regulator of ROS escape from mitochondria.

AB - Reactive oxygen species (ROS) are generated during T cell activation and serve a signaling function but can also be damaging. In this issue of Immunity, Zhang et al. (2016) show that miR-23a prevents ROS-elicited necrosis by suppressing cyclophilin D (PPIF), a regulator of ROS escape from mitochondria.

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U2 - 10.1016/j.immuni.2016.02.027

DO - 10.1016/j.immuni.2016.02.027

M3 - Short survey

C2 - 26982360

AN - SCOPUS:84960390820

SN - 1074-7613

VL - 44

SP - 520

EP - 522

JO - Immunity

JF - Immunity

IS - 3

ER -

Silence of the ROS

Abstract

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