Role of the Complement Pathway in Inflammatory Skin Diseases: A Focus on Hidradenitis Suppurativa

Mondana H. Ghias, Michael J. Hyde, Lewis E. Tomalin, B. Paul Morgan, Afsaneh Alavi, Michelle A. Lowes, Vincent Piguet

Research output: Contribution to journalReview articlepeer-review

27 Scopus citations

Abstract

Although the role of immune dysregulation in hidradenitis suppurativa (HS) has yet to be elucidated, recent studies identified several complement abnormalities in patients with HS. The complement system serves a critical role in the modulation of immune response and regulation of cutaneous commensal bacteria. Complement is implicated in several inflammatory skin diseases including systemic lupus erythematosus, angioedema, pemphigus, bullous pemphigoid, and HS. A model of HS pathogenesis is proposed, integrating the role of commensal bacteria, cutaneous immune responses, and complement dysregulation. The role of complement in disease pathogenesis has led to the development of novel anticomplement agents and clinical trials investigating the efficacy of such treatments in HS.

Original languageEnglish
Pages (from-to)531-536.e1
JournalJournal of Investigative Dermatology
Volume140
Issue number3
DOIs
StatePublished - Mar 2020

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