Response to comments on “Aberrant type 1 immunity drives susceptibility to mucosal fungal infections”

Genomics and Computational Biology Core

Research output: Contribution to journalArticlepeer-review

4 Scopus citations

Abstract

Puel and Casanova and Kisand et al. challenge our conclusions that interferonopathy and not IL-17/IL-22 autoantibodies promote candidiasis in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy. We acknowledge that conclusive evidence for causation is difficult to obtain in complex human diseases. However, our studies clearly document interferonopathy driving mucosal candidiasis with intact IL-17/IL-22 responses in Aire-deficient mice, with strong corroborative evidence in patients.

Original languageEnglish
JournalScience
Volume373
Issue number6561
DOIs
StatePublished - 17 Sep 2021

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