Regulation of Fas expression by STAT3 and c-Jun is mediated by phosphatidylinositol 3-kinase-AKT signaling

Keyphrases

• C-Jun 100%
• Phosphoinositide 3-kinase (PI3K) 100%
• AKT Pathway 100%
• Fas Expression 100%
• Fas Receptor 100%
• Receptor Expression 20%
• Protein Kinase 10%
• Fibroblasts 10%
• Transcription Factor 10%
• Pharmacological Inhibitors 10%
• Phosphorylation 10%
• Human Tumors 10%
• Transcriptional Activity 10%
• Programmed Cell Death 10%
• FasL 10%
• Negative Effects 10%
• Dominant Negative 10%
• Surface Expression 10%
• Promoter Activity 10%
• LY294002 10%
• Negative Type 10%
• Derepression 10%
• JNK Activation 10%
• Phosphatidylinositol 3-kinase/AKT Signaling Pathway 10%

Medicine and Dentistry

• Phosphoinositide 3-Kinase 100%
• Tumor Necrosis Factor Receptor Superfamily Member 6 100%
• Cooperation 30%
• Promoter Region 20%
• Receptor Expression 20%
• Neoplasm 10%
• Transcription Factors 10%
• Programmed Cell Death 10%
• Signal Transduction 10%
• Sensitization 10%
• Surface Receptor 10%
• LY294002 10%
• Protein Kinases 10%
• Fibroblast 10%

Pharmacology, Toxicology and Pharmaceutical Science

• Phosphatidylinositol 3 Kinase 100%
• Tumor Necrosis Factor Receptor Superfamily Member 6 100%
• Neoplasm 10%
• Transcription Factors 10%
• Programmed Cell Death 10%
• Protein Kinases 10%
• 2 Morpholino 8 Phenylchromone 10%