Regulated complement deposition on the surface of human endothelial cells: Effect of tobacco smoke and shear stress

Keyphrases

• Endothelial Cells 100%
• Tobacco Smoke 100%
• Shear Stress 100%
• Cell Effects 100%
• Complement Deposition 100%
• Human Endothelial Cells 100%
• Complement Activation 60%
• SC5b-9 60%
• IC3b 60%
• Cell Surface 40%
• Lipopolysaccharide 40%
• Endothelial Monolayer 40%
• Bone Marrow 20%
• Direct Activation 20%
• Endothelial Cell Activation 20%
• Vascular Inflammation 20%
• Thrombosis 20%
• Human Plasma 20%
• Cigarette Smoking 20%
• Cell-mediated 20%
• In Situ 20%
• Complement C3 20%
• Surface Expression 20%
• Complement Regulatory Proteins 20%
• Complement Regulation 20%
• Alternative Pathway 20%
• Vascular Endothelial Cells 20%
• Arteriosclerosis 20%
• Vascular Injury 20%
• Hemodynamic Stress 20%
• Atherogenesis 20%
• Human Umbilical Vein Endothelial Cells (HUVECs) 20%
• C3 Convertase 20%
• CD55 20%
• Classical Pathway 20%
• C4d Deposition 20%
• Endothelial Cell Injury 20%
• Surface Deposition 20%
• Complement Component C4 20%
• Tobacco Smoke Extract 20%
• Cell Exposure 20%
• Exposure to Tobacco Smoke 20%
• CD35 20%
• ICAM-1 Expression 20%

Immunology and Microbiology

• Shear Stress 100%
• Complement Deposition 100%
• Complement Component C4d 80%
• Complement Activation 60%
• IC3b 60%
• Blood Plasma 40%
• Cell Surface 40%
• Atherosclerosis 20%
• Cell Damage 20%
• Cell Activation 20%
• Vasculitis 20%
• Mediator 20%
• ELISA 20%
• Decay-Accelerating Factor 20%
• Complement Component 3 20%
• Complement Factor B 20%
• Hemodynamic Stress 20%
• Intercellular Adhesion Molecule 1 20%
• Umbilical Vein Endothelial Cell 20%
• Complement Component 4 20%