Primed CD8 ⁺ T-cell responses to allogeneic endothelial cells are controlled by local complement activation

Keyphrases

• Endothelial Cells 100%
• Cell Response 100%
• Allogeneic 100%
• CD8+ T 100%
• Complement Activation 100%
• Local Complementation 100%
• CD8+ T Cells 60%
• C5a Receptor 60%
• T Cells 40%
• Cell-derived 40%
• Decay Accelerating Factor 40%
• Transplantation 20%
• Immune Cells 20%
• Inflammatory Cytokines 20%
• T Cell Activation 20%
• Recombinant 20%
• Cell Surface 20%
• Cell Interaction 20%
• T-cell Mediated 20%
• Abrogation 20%
• T Cell Proliferation 20%
• CD-8 20%
• Receptor-ligand Interaction 20%
• Effector Cells 20%
• T Cell Expansion 20%
• Alternative Pathway 20%
• Vascular Endothelium 20%
• MHC Class I 20%
• Receptor Blockade 20%
• Allograft Injury 20%
• Perforin 20%
• Graft Injury 20%
• MHC Molecules 20%
• CD55 20%
• C5 Convertase 20%
• Complement Activation Fragments 20%
• Cell Decay 20%
• Transplanted Heart 20%
• CD8 T Cell Differentiation 20%

Immunology and Microbiology

• Cytotoxic T-Cell 100%
• Complement Activation 100%
• T Cell 75%
• Complement Component C5a Receptor 75%
• Decay-Accelerating Factor 75%
• CD8 50%
• Complement Component 5a 50%
• Immunocompetent Cell 25%
• Wild Type 25%
• T Cell Activation 25%
• Proinflammatory Cytokine 25%
• Cell Surface 25%
• Cell Interaction 25%
• Cell Differentiation 25%
• Effector Cell 25%
• T Cell Proliferation 25%
• Alternative Complement Pathway 25%
• Cell Expansion 25%
• MHC Class I 25%
• Vascular Endothelium 25%
• Receptor Blocking 25%
• Perforin 25%
• C5-Convertase 25%
• Allograft 25%