POSITRON EMISSION TOMOGRAPHIC STUDIES ON INDUCED MIGRAINE

H. Sachs; J. A.G. Russell; D. R. Christman; J. S. Fowler; A. P. Wolf

doi:10.1016/S0140-6736(84)92940-4

POSITRON EMISSION TOMOGRAPHIC STUDIES ON INDUCED MIGRAINE

H. Sachs
, J. A.G. Russell
, D. R. Christman
, J. S. Fowler
, A. P. Wolf

Research output: Contribution to journal › Letter › peer-review

2 Scopus citations

Original language	English
Pages (from-to)	465
Number of pages	1
Journal	The Lancet
Volume	324
Issue number	8400
DOIs	https://doi.org/10.1016/S0140-6736(84)92940-4
State	Published - 25 Aug 1984
Externally published	Yes

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10.1016/S0140-6736(84)92940-4

Cite this

@article{592491d864a146b8b9b2356547a356c9,

title = "POSITRON EMISSION TOMOGRAPHIC STUDIES ON INDUCED MIGRAINE",

author = "H. Sachs and Russell, \{J. A.G.\} and Christman, \{D. R.\} and Fowler, \{J. S.\} and Wolf, \{A. P.\}",

note = "Funding Information: It would thus appear, in these preliminary experiments, that cerebral glucose hypometabolism is an initial event in the migrainous headache induced by reserpine in susceptible subjects. The observed decline in RCMRGlu amounts to a considerable impairment in cerebral metabolism and would be consistent with the model of deranged cerebral function proposed by Pearce, Edmeads,l and others2 as the primary event in the genesis of the migraine attack. This research was carried out under contract DE-AC02-76CH00016 with the US Department of Energy and supported by its Office of Health and Environmental Research, and also suooorted bv National Institutes of Health",

year = "1984",

month = aug,

day = "25",

doi = "10.1016/S0140-6736(84)92940-4",

language = "English",

volume = "324",

pages = "465",

journal = "The Lancet",

issn = "0140-6736",

publisher = "Elsevier B.V.",

number = "8400",

}

TY - JOUR

T1 - POSITRON EMISSION TOMOGRAPHIC STUDIES ON INDUCED MIGRAINE

AU - Sachs, H.

AU - Russell, J. A.G.

AU - Christman, D. R.

AU - Fowler, J. S.

AU - Wolf, A. P.

N1 - Funding Information: It would thus appear, in these preliminary experiments, that cerebral glucose hypometabolism is an initial event in the migrainous headache induced by reserpine in susceptible subjects. The observed decline in RCMRGlu amounts to a considerable impairment in cerebral metabolism and would be consistent with the model of deranged cerebral function proposed by Pearce, Edmeads,l and others2 as the primary event in the genesis of the migraine attack. This research was carried out under contract DE-AC02-76CH00016 with the US Department of Energy and supported by its Office of Health and Environmental Research, and also suooorted bv National Institutes of Health

PY - 1984/8/25

Y1 - 1984/8/25

UR - https://www.scopus.com/pages/publications/0021173191

U2 - 10.1016/S0140-6736(84)92940-4

DO - 10.1016/S0140-6736(84)92940-4

M3 - Letter

C2 - 6147532

AN - SCOPUS:0021173191

SN - 0140-6736

VL - 324

SP - 465

JO - The Lancet

JF - The Lancet

IS - 8400

ER -