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Pathophysiology of coronary artery disease
A. Fernandez-Ortiz
,
V. Fuster
Artificial Intelligence and Human Health
BioMedical Engineering and Imaging Institute
Friedman Brain Institute
Icahn School of Medicine at Mount Sinai
Medicine
Medicine - Cardiology
Research output
:
Contribution to journal
›
Review article
›
peer-review
10
Scopus citations
Overview
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Keyphrases
Coronary Artery Disease
100%
Pathophysiology
100%
Thrombus Formation
100%
Atherosclerotic Lesions
66%
Plaque Rupture
66%
Endothelium
66%
Lipid Accumulation
66%
Smooth muscle Cell Proliferation
66%
Risk Factors
33%
Acute Coronary Syndrome
33%
Clinically Significant
33%
Macrophages
33%
Plaque
33%
Chronic Injury
33%
Arterial Tree
33%
Lipid-rich
33%
Extracellular Lipids
33%
Slow Progression
33%
Clinically Silent
33%
Collagen Matrix
33%
Fissure
33%
Matrix Synthesis
33%
Sclerotic Lesion
33%
Atherosclerotic Process
33%
Mural Thrombus
33%
Atherogenic Risk
33%
Massive Thrombus
33%
Silent Progression
33%
Medicine and Dentistry
Coronary Artery Disease
100%
Pathophysiology
100%
Blood Clotting
100%
Lipid Storage
66%
Cell Proliferation
66%
Smooth Muscle Cell
66%
Macrophage
33%
Arterial Tree
33%
Acute Coronary Syndrome
33%