Oncogenesis by E2A-PBX1 in ALL: RUNX and more

Jonathan D. Licht

Research output: Contribution to journalReview articlepeer-review

3 Scopus citations

Abstract

The mechanisms by which fusion transcription factors stimulate cancer pathogenesis are being elucidated with increasingly powerful molecular techniques. In this issue of Blood, Pi et al1 performed a genome-wide identification of the targets of the E2A-PBX1 fusion protein associated with t(1;19), found in 4% of cases of childhood B-cell acute lymphocytic leukemia (ALL).

Original languageEnglish
Pages (from-to)3-4
Number of pages2
JournalBlood
Volume136
Issue number1
DOIs
StatePublished - Jul 2020
Externally publishedYes

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