Neurobiology of anxiety disorders and implications for treatment

Keyphrases

• Implications for Treatment 100%
• Anxiety Disorders 100%
• Amygdala 100%
• Neurobiology 100%
• Conditioned Stimulus 100%
• Posttraumatic Stress Disorder 66%
• Fear Conditioning 66%
• Pharmacological Manipulation 33%
• Psychotherapy 33%
• Beta-blockers 33%
• Contextual Fear Conditioning 33%
• Panic Disorder 33%
• Propranolol 33%
• β-adrenergic Antagonist 33%
• Cognitive Behavioral Therapy 33%
• Voltage-gated Calcium Channels 33%
• N-methyl-D-aspartate 33%
• Anxiety Treatment 33%
• Anti-anxiety 33%
• Classical Conditioning 33%
• Glutamate 33%
• Specific Phobia 33%
• N-methyl-D-aspartate Receptor Antagonist 33%
• Fear Extinction 33%
• D-cycloserine 33%
• Aversive Stimuli 33%
• New Learning 33%
• Traumatic Memory 33%
• Footshock 33%
• Fear Inhibition 33%
• Pavlovian Conditioning 33%
• Good Durability 33%
• Fear Circuit 33%

Neuroscience

• Anxiety Disorder 100%
• Amygdala 100%
• Neurobiology 100%
• Posttraumatic Stress Disorder 66%
• Aspartic Acid 66%
• Fear Conditioning 66%
• Classical Conditioning 66%
• Agonist 33%
• Panic Disorder 33%
• Adrenergic Beta-Antagonist 33%
• Cognitive Behavioral Therapy 33%
• Glutamic Acid 33%
• Cycloserine 33%
• Voltage Gated Calcium Channel 33%
• Propranolol 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Anxiety Disorder 100%
• Aspartic Acid 66%
• Posttraumatic Stress Disorder 66%
• Beta Adrenergic Receptor Blocking Agent 33%
• Propranolol 33%
• Panic 33%
• Glutamic Acid 33%
• Preclinical Study 33%
• Cycloserine 33%
• Voltage Gated Calcium Channel 33%