Nanoformulation of PROteolysis TArgeting Chimera targeting ‘undruggable’ c-Myc for the treatment of pancreatic cancer

  • Aishwarya Saraswat
  • , Manali Patki
  • , Yige Fu
  • , Shrikant Barot
  • , Vikas V. Dukhande
  • , Ketan Patel

Research output: Contribution to journalArticlepeer-review

85 Scopus citations

Abstract

Aim: To explore the anticancer activity of a novel BRD4 protein degrader ARV-825 (ARV) and its nanoformulation development (ARV-NP) for treatment of pancreatic cancer. Materials & methods: ARV-NP were prepared using nanoprecipitation method and characterized for their physicochemical properties and various anticancer cell culture assays. Results: ARV-NP (89.63 ± 16.39 nm) demonstrated good physical stability, negligible hemolysis and improved half-life of ARV. ARV-NP showed significant cytotoxicity, apoptosis and anticlonogenic effect in pancreatic cancer cells. Significant downregulation of target proteins BRD4, c-Myc, Bcl-2 and upregulation of apoptotic marker cleaved caspase-3 was observed. Most importantly, ARV-NP treatment significantly inhibited the cell viability of 3D tumor spheroids of pancreatic cancer. Conclusion: ARV-NP represents a novel therapeutic strategy for pancreatic cancer.

Original languageEnglish
Pages (from-to)1761-1777
Number of pages17
JournalNanomedicine
Volume15
Issue number18
DOIs
StatePublished - Aug 2020
Externally publishedYes

Keywords

  • ARV-825
  • BRD4
  • Drug delivery
  • PROTAC
  • Pancreatic ductal adenocarcinoma
  • Polymeric nanoparticles

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