Abstract

The MYC oncogene is a multifunctional protein that is aberrantly expressed in a significant fraction of tumors from diverse tissue origins. Because of its multifunctional nature, it has been difficult to delineate the exact contributions of MYC’s diverse roles to tumorigenesis. Here, we review the normal role of MYC in regulating DNA replication as well as its ability to generate DNA replication stress when overexpressed. Finally, we discuss the possible mech- anisms by which replication stress induced by aberrant MYC expression could contribute to genomic instability and cancer.

Original languageEnglish
JournalCold Spring Harbor Perspectives in Medicine
Volume4
Issue number6
DOIs
StatePublished - 2014

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