Interaction of prenatal exposure to cigarettes and MAOA genotype in pathways to youth antisocial behavior

Keyphrases

• Genotype 100%
• Prenatal Exposure 100%
• Cigarette 100%
• Monoamine Oxidase A (MAO-A) 100%
• Youth Antisocial Behavior 100%
• Variable number of Tandem Repeats 60%
• Information Processing 40%
• 3′-untranslated Region (3′-UTR) 40%
• Conduct Disorder Symptoms 40%
• Sex-specific Patterns 20%
• Brain Function 20%
• Genetic Susceptibility 20%
• 5′-untranslated Region (5′-UTR) 20%
• Low Activity 20%
• Functional Polymorphism 20%
• High Activity 20%
• Exposure to Cigarette Smoke 20%
• Pregnancy Cohort 20%
• Precise Measurement 20%
• Face Processing 20%
• Gene-environment Correlation 20%
• Hostile Attribution Bias 20%
• Putative Genes 20%
• Processing Patterns 20%
• Exposure Mechanism 20%
• Symptom Attribution 20%

Neuroscience

• Behavior (Neuroscience) 100%
• Monoamine Oxidase A 100%
• Untranslated Region 60%
• Variable Number Tandem Repeat 60%
• Information Processing 40%
• Conduct Disorder 40%
• Brain Function 20%
• Face 20%

Psychology

• Antisocial Behavior 100%
• Conduct Disorder 50%
• Information Processing 50%
• Adolescence 50%