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Holoprosencephaly: New models, new insights
Robert S. Krauss
Black Family Stem Cell Institute
Cancer Mechanisms
Cell, Developmental, and Regenerative Biology
Environmental Medicine and Climate Science
Friedman Brain Institute
Graduate School of Biomedical Sciences
Icahn School of Medicine at Mount Sinai
Institute for Climate Change, Environmental Health, and Exposomics
Mindich Child Health and Development Institute
The Tisch Cancer Institute
Research output
:
Contribution to journal
›
Review article
›
peer-review
48
Scopus citations
Overview
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Keyphrases
Holoprosencephaly
100%
Familial mutations
40%
Genetic Variation
20%
Forebrain
20%
Environmental Factors
20%
Clinical Presentation
20%
Signal Transduction Pathway
20%
Left Hemisphere
20%
Right Hemisphere
20%
Autosomal Dominant
20%
Complex Interactions
20%
Congenital Malformations
20%
Structural Anomalies
20%
Sporadic Form
20%
Environmental Agents
20%
Family Forms
20%
Obligate Carrier
20%
New Animal Model
20%
Developmental Signaling Pathways
20%
Body Structure
20%
Biochemistry, Genetics and Molecular Biology
Signal Transduction
100%
Animal Model
50%
Genetic Divergence
50%
Genetic Variation
50%
Autosomal Dominant Inheritance
50%
Obligate Carrier
50%
Medicine and Dentistry
Holoprosencephaly
100%
Congenital Malformation
20%
Signal Transduction
20%
Signal Transduction Pathway
20%
Autosomal Dominant Inheritance
20%
Environmental Factor
20%
Genetic Variation
20%
Right Hemisphere
20%
Left Hemisphere
20%
Body Structure
20%
Obligate Carrier
20%
Pharmacology, Toxicology and Pharmaceutical Science
Holoprosencephaly
100%
Congenital Malformation
20%