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Hepatic inflammatory responses in liver fibrosis
Linda Hammerich, Frank Tacke
Research output
:
Contribution to journal
›
Review article
›
peer-review
469
Scopus citations
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Dive into the research topics of 'Hepatic inflammatory responses in liver fibrosis'. Together they form a unique fingerprint.
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Keyphrases
Inflammatory Response
100%
Liver Fibrosis
100%
Non-alcoholic Fatty Liver Disease (NAFLD)
100%
Nonalcoholic Steatohepatitis
66%
Inflammation
66%
Liver
66%
Viral Hepatitis
33%
Unmet Medical Need
33%
Therapeutic Target
33%
T Cells
33%
Fibrosis
33%
Cirrhosis
33%
Inflammatory Cells
33%
Chemokines
33%
Neutrophils
33%
Liver Cancer
33%
Inflammatory Pathways
33%
Therapeutic Implications
33%
Disease States
33%
Chronic Liver Disease
33%
Hepatic Stellate Cells
33%
Immune Activation
33%
Inflammatory Mediators
33%
Hepatic Stellate Cell Activation
33%
Fibrogenic
33%
Hepatic Cells
33%
Cell Subsets
33%
Innate Lymphoid Cells
33%
Tissue Repair
33%
Persistent Inflammation
33%
T Cell Subpopulations
33%
Immune Defense
33%
Transdifferentiate
33%
Diseased Liver
33%
Death Signals
33%
Spatial Organization
33%
Long-term Morbidity
33%
Myofibroblast
33%
Liver-related Mortality
33%
Resident Macrophages
33%
Matrix Producing
33%
Recruited Macrophages
33%
Single-cell Technologies
33%
Aggressive Features
33%
Unconventional T Cells
33%
Hepatocellular Death
33%
Medicine and Dentistry
Nonalcoholic Fatty Liver
100%
Serositis
100%
Liver Fibrosis
100%
Mediator
40%
Hepatic Stellate Cell
40%
T Cell
40%
Inflammatory Cell
20%
Cytokine
20%
Immunocompetent Cell
20%
Cell Population
20%
Cell Activation
20%
Liver Cirrhosis
20%
Fibrosis
20%
Macrophage
20%
Liver Cancer
20%
Chemokine
20%
Chronic Liver Disease
20%
Hepatitis Virus
20%
Fibrogenesis
20%
Inflammatory Mediator
20%
Tissue Repair
20%
Lymphoid Cell
20%
Diseases
20%
Myofibroblast
20%
Neutrophil
20%