Skip to main navigation
Skip to search
Skip to main content
Icahn School of Medicine at Mount Sinai Home
Help & FAQ
Link opens in a new tab
Search content at Icahn School of Medicine at Mount Sinai
Home
Profiles
Research units
Publications & Research Outputs
Press/Media
G
q
signaling in α cells is critical for maintaining euglycemia
Liu Liu
, Diptadip Dattaroy
, Katherine F. Simpson
, Luiz F. Barella
, Yinghong Cui
, Yan Xiong
,
Jian Jin
, Gabriele M. König
, Evi Kostenis
, Jefferey C. Roman
, Klaus H. Kaestner
, Nicolai M. Doliba
, Jürgen Wess
Graduate School of Biomedical Sciences
Icahn School of Medicine at Mount Sinai
Cancer Clinical Investigation
Friedman Brain Institute
Mount Sinai Tisch Cancer Center
Oncological Sciences
Pharmacological Sciences
Research output
:
Contribution to journal
›
Article
›
peer-review
21
Scopus citations
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'G
q
signaling in α cells is critical for maintaining euglycemia'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Keyphrases
Euglycemia
100%
Gq Signaling
100%
Glucose Tolerance
50%
Glucagon
50%
Mouse Cells
50%
Glucagon Secretion
50%
Gq-coupled Receptor
50%
Glucagon Release
50%
Human Cells
25%
G Protein
25%
Pancreatic Cells
25%
Novel Therapeutics
25%
Induced Activation
25%
Signaling Pathway
25%
Metabolic Effects
25%
Obese Mice
25%
Hyperglucagonemia
25%
Regulation of Cell Functions
25%
Insulin Release
25%
Potential Regulation
25%
Vasopressin 1b Receptor
25%
Acute Stimulation
25%
Chemogenetics
25%
Biochemistry, Genetics and Molecular Biology
Glucagon Release
100%
Glucose
50%
Signal Transduction
25%
Cell Function
25%
Agonist
25%
Mouse Mutant
25%
Insulin Release
25%
Chemogenetics
25%
Glucagon Blood Level
25%
In Vivo Study
25%
Vasopressin Receptor
25%
G Protein
25%
Glucagon
25%
Neuroscience
Glucagon
100%
In Vivo
33%
Receptor
33%
Glucose
33%
G Protein
16%
In Vitro
16%
Agonist
16%
Cell Signaling
16%
Cell Function
16%
Insulin
16%
Vasopressin V1b Receptor
16%
Blood Plasma
16%
Pharmacology, Toxicology and Pharmaceutical Science
Glucagon
100%
Receptor
33%
Glucose
33%
Mouse Mutant
16%
Guanine Nucleotide Binding Protein
16%
Antidiabetic Agent
16%
Insulin
16%
Hyperglucagonemia
16%
Vasopressin V1b Receptor
16%