Glatiramer acetate-reactive T lymphocytes regulate oligodendrocyte progenitor cell number in vitro: Role of IGF-2

Y. Zhang, F. Jalili, N. Ouamara, A. Zameer, G. Cosentino, M. Mayne, L. Hayardeny, J. P. Antel, A. Bar-Or, G. R. John

Research output: Contribution to journalArticlepeer-review

31 Scopus citations

Abstract

Glatiramer acetate (GA) is an immunomodulator approved for therapy of relapsing-remitting multiple sclerosis (RRMS), but recent findings indicate that it may also have additional, neurotrophic effects. Here, we found that supernatants from human GA-reactive T lymphocytes potentiated oligodendrocyte numbers in rodent and human oligodendrocyte progenitor (OPC) cultures. Effects of Th2-polarized lines were stronger than Th1-polarized cells. Microarray and ELISA analyses revealed that neurotrophic factors induced in Th2- and Th1-polarized GA-reactive lines included IGF-2 and BMP-7 respectively, and functional studies confirmed IGF-2 as trophic for OPCs. Our results support the concept that GA therapy may result in supportive effects on oligodendrocytes in RRMS patients.

Original languageEnglish
Pages (from-to)71-79
Number of pages9
JournalJournal of Neuroimmunology
Volume227
Issue number1-2
DOIs
StatePublished - Oct 2010
Externally publishedYes

Keywords

  • CNS repair
  • Glatiramer acetate
  • Multiple sclerosis
  • Oligodendrocyte progenitor
  • T lymphocyte

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