Genetic architecture distinguishes tinnitus from hearing loss

Royce E. Clifford, Adam X. Maihofer, Chris Chatzinakos, Jonathan R.I. Coleman, Nikolaos P. Daskalakis, Marianna Gasperi, Kelleigh Hogan, Elizabeth A. Mikita, Murray B. Stein, Catherine Tcheandjieu, Francesca Telese, Yanning Zuo, Allen F. Ryan, Caroline M. Nievergelt

Research output: Contribution to journalArticlepeer-review

3 Scopus citations

Abstract

Tinnitus is a heritable, highly prevalent auditory disorder treated by multiple medical specialties. Previous GWAS indicated high genetic correlations between tinnitus and hearing loss, with little indication of differentiating signals. We present a GWAS meta-analysis, triple previous sample sizes, and expand to non-European ancestries. GWAS in 596,905 Million Veteran Program subjects identified 39 tinnitus loci, and identified genes related to neuronal synapses and cochlear structural support. Applying state-of-the-art analytic tools, we confirm a large number of shared variants, but also a distinct genetic architecture of tinnitus, with higher polygenicity and large proportion of variants not shared with hearing difficulty. Tissue-expression analysis for tinnitus infers broad enrichment across most brain tissues, in contrast to hearing difficulty. Finally, tinnitus is not only correlated with hearing loss, but also with a spectrum of psychiatric disorders, providing potential new avenues for treatment. This study establishes tinnitus as a distinct disorder separate from hearing difficulties.

Original languageEnglish
Article number614
JournalNature Communications
Volume15
Issue number1
DOIs
StatePublished - Dec 2024
Externally publishedYes

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