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Fibroblast Growth Factor 15/19 in Hepatocarcinogenesis
Gloria Alvarez-Sola
, Iker Uriarte
, M. Ujue Latasa
, Raquel Urtasun
, Marina Bárcena-Varela
, Maria Elizalde
, Maddalen Jiménez
, Carlos M. Rodriguez-Ortigosa
, Fernando J. Corrales
, Maite G. Fernández-Barrena
, Carmen Berasain
, Matías A. Avila
Research output
:
Contribution to journal
›
Article
›
peer-review
36
Scopus citations
Overview
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Keyphrases
FGF Receptor
100%
Hepatocarcinogenesis
100%
Fibroblast Growth Factor 19 (FGF19)
100%
Fgf15
88%
Hepatocellular Carcinoma Patients
22%
Signaling System
22%
Tumoral
22%
Bile Acid Metabolism
22%
Hepatocellular Carcinoma
11%
Molecular Mechanism
11%
Tumor
11%
Overexpression
11%
Conventional Chemotherapy
11%
Targeted Therapy
11%
Worse Prognosis
11%
Poor Prognosis
11%
Clinical Efficacy
11%
Advanced Hepatocellular Carcinoma
11%
Patient-centered
11%
Sorafenib
11%
Poor Performance
11%
Heterogeneous Tumors
11%
Toxic Effects
11%
Hepatocellular Carcinoma Cells
11%
Aberrant Signaling
11%
Molecular Subclass
11%
Intermediary Metabolism
11%
Targeted Drugs
11%
Multikinase Inhibitor
11%
Hepatocellular Carcinoma Progression
11%
Neoplastic Disease
11%
Klotho
11%
Hepatocellular Carcinoma Therapy
11%
Experimental Hepatocarcinogenesis
11%
Pharmacological Interferences
11%
Medicine and Dentistry
Fibroblast Growth Factor
100%
Liver Carcinogenesis
100%
Fibroblast Growth Factor Receptor 4
100%
Hepatocellular Carcinoma
77%
Fibroblast Growth Factor 19
33%
Tumor
22%
Neoplasm
11%
Biological Marker
11%
Targeted Therapy
11%
Carcinoma Cell
11%
Sorafenib
11%
Bile Acid Synthesis
11%
Bile Acid Metabolism
11%
Pharmacology, Toxicology and Pharmaceutical Science
Fibroblast Growth Factor
100%
Liver Carcinogenesis
100%
Fibroblast Growth Factor Receptor 4
100%
Liver Cell Carcinoma
77%
Fibroblast Growth Factor 19
33%
Bile Acid
22%
Neoplasm
11%
Biological Marker
11%
Chemotherapy
11%
Sorafenib
11%