Feline models of mucopolysaccharidosis

M. E. Haskins; P. F. Jezyk; R. J. Desnick; D. F. Patterson

Feline models of mucopolysaccharidosis

M. E. Haskins
, P. F. Jezyk
, R. J. Desnick
, D. F. Patterson

Research output: Contribution to journal › Article › peer-review

3 Scopus citations

Abstract

The potential value of animal models for the investigation of the pathogenesis and approaches to therapy in human disease is well recognized. A number of counterparts of human lysosomal storage diseases have been reported in domestic animals, including G(M1) gangliosidosis (cat), G(M2) gangliosidosis (cat, dog, pig), globoid cell leukodystrophy (dog) mannosidosis (cattle), and glycogenosis (cattle). The first animal models of the genetic mucopolysaccharidoses (I and VI), recently reported by us in domestic cats are the subject of this brief communication.

Original language	English
Pages (from-to)	219-224
Number of pages	6
Journal	Birth Defects: Original Article Series
Volume	17
Issue number	1
State	Published - 1981
Externally published	Yes

Cite this

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abstract = "The potential value of animal models for the investigation of the pathogenesis and approaches to therapy in human disease is well recognized. A number of counterparts of human lysosomal storage diseases have been reported in domestic animals, including G(M1) gangliosidosis (cat), G(M2) gangliosidosis (cat, dog, pig), globoid cell leukodystrophy (dog) mannosidosis (cattle), and glycogenosis (cattle). The first animal models of the genetic mucopolysaccharidoses (I and VI), recently reported by us in domestic cats are the subject of this brief communication.",

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year = "1981",

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journal = "Birth Defects: Original Article Series",

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AU - Haskins, M. E.

AU - Jezyk, P. F.

AU - Desnick, R. J.

AU - Patterson, D. F.

PY - 1981

Y1 - 1981

N2 - The potential value of animal models for the investigation of the pathogenesis and approaches to therapy in human disease is well recognized. A number of counterparts of human lysosomal storage diseases have been reported in domestic animals, including G(M1) gangliosidosis (cat), G(M2) gangliosidosis (cat, dog, pig), globoid cell leukodystrophy (dog) mannosidosis (cattle), and glycogenosis (cattle). The first animal models of the genetic mucopolysaccharidoses (I and VI), recently reported by us in domestic cats are the subject of this brief communication.

AB - The potential value of animal models for the investigation of the pathogenesis and approaches to therapy in human disease is well recognized. A number of counterparts of human lysosomal storage diseases have been reported in domestic animals, including G(M1) gangliosidosis (cat), G(M2) gangliosidosis (cat, dog, pig), globoid cell leukodystrophy (dog) mannosidosis (cattle), and glycogenosis (cattle). The first animal models of the genetic mucopolysaccharidoses (I and VI), recently reported by us in domestic cats are the subject of this brief communication.

UR - https://www.scopus.com/pages/publications/0019126585

M3 - Article

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AN - SCOPUS:0019126585

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EP - 224

JO - Birth Defects: Original Article Series

JF - Birth Defects: Original Article Series

IS - 1

ER -

Feline models of mucopolysaccharidosis

Abstract

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