Abstract
The potential value of animal models for the investigation of the pathogenesis and approaches to therapy in human disease is well recognized. A number of counterparts of human lysosomal storage diseases have been reported in domestic animals, including G(M1) gangliosidosis (cat), G(M2) gangliosidosis (cat, dog, pig), globoid cell leukodystrophy (dog) mannosidosis (cattle), and glycogenosis (cattle). The first animal models of the genetic mucopolysaccharidoses (I and VI), recently reported by us in domestic cats are the subject of this brief communication.
| Original language | English |
|---|---|
| Pages (from-to) | 219-224 |
| Number of pages | 6 |
| Journal | Birth Defects: Original Article Series |
| Volume | 17 |
| Issue number | 1 |
| State | Published - 1981 |
| Externally published | Yes |