Effect of phagocytosis and lonophores on release and metabolism of arachidonic acid from human neutrophils

Christopher E. Walsh, Lawrence R. Dechatelet, Michael J. Thomas, Joseph T. O'Flaherty, Moseley Waite

Research output: Contribution to journalArticlepeer-review

36 Scopus citations

Abstract

Challenge of human neutrophils prelabeled with [3H]arachidonate and [14C] palmitate or [14C]-stearate with opsonized zymosan or the Ca2+ ionophores A23187 or Ionomycin caused the release of [3H], but not [14C], fatty acid. With the ionophores, but not zymosan, considerable conversion of the [3H] arachidonate to hydroxyeicosatetraenoates occurred. Although various isomers were recovered, the 5-hydroxyeicosatetraenoate appeared to be the major product. In these experiments, no [14C] products were detected such as lysophospholipid, diglyceride or monoglyceride. Although no definitive statement can be made about the mechanism of release of arachidonate, our data are most easily interpreted as the result of the action of a phospholipase A2.

Original languageEnglish
Pages (from-to)120-124
Number of pages5
JournalLipids
Volume16
Issue number2
DOIs
StatePublished - Feb 1981
Externally publishedYes

Fingerprint

Dive into the research topics of 'Effect of phagocytosis and lonophores on release and metabolism of arachidonic acid from human neutrophils'. Together they form a unique fingerprint.

Cite this