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Desmoid fibromatosis is a clonal process
Maomi Li
, Carlos Cordon-Cardo
, William L. Gerald
, Juan Rosai
Research output
:
Contribution to journal
›
Article
›
peer-review
152
Scopus citations
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Keyphrases
Primary Lesion
100%
Desmoid-type Fibromatosis
100%
Recurrent Lesions
66%
HUMARA
66%
X-inactivation
66%
Methylation Status
33%
Polymorphic Site
33%
Androgen Receptor
33%
Female Patients
33%
X Chromosome
33%
Cell Clones
33%
DNA Methylation Pattern
33%
Lesional
33%
Digestion
33%
Clonality
33%
Molecular Genetic Analysis
33%
Locally Aggressive
33%
HpaII
33%
Incorrect Conclusions
33%
Clonal Origin
33%
Soft Tissue Lesions
33%
Biochemistry, Genetics and Molecular Biology
X-Inactivation
100%
Methylation
50%
DNA Methylation
50%
Androgen Receptor
50%
Cell Clone
50%
X Chromosome
50%
Molecular Genetics
50%
Clonality
50%
Clonal Variation
50%