Designing a Type I Interferon Signaling Phagosome

J. Magarian Blander

doi:10.1016/j.immuni.2012.11.009

Designing a Type I Interferon Signaling Phagosome

J. Magarian Blander

Research output: Contribution to journal › Short survey › peer-review

3 Scopus citations

Abstract

Circulating immune complexes trigger type I interferons exacerbating disease in systemic lupus erythematosus. In this issue of Immunity, Henault et al. (2012) show that Fcγ-receptor, Toll-like receptor 9, and LC3 conspire to mold phagosomes into type I interferon signaling platforms.

Original language	English
Pages (from-to)	947-949
Number of pages	3
Journal	Immunity
Volume	37
Issue number	6
DOIs	https://doi.org/10.1016/j.immuni.2012.11.009
State	Published - 14 Dec 2012

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title = "Designing a Type I Interferon Signaling Phagosome",

abstract = "Circulating immune complexes trigger type I interferons exacerbating disease in systemic lupus erythematosus. In this issue of Immunity, Henault et al. (2012) show that Fcγ-receptor, Toll-like receptor 9, and LC3 conspire to mold phagosomes into type I interferon signaling platforms.",

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doi = "10.1016/j.immuni.2012.11.009",

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AU - Blander, J. Magarian

PY - 2012/12/14

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AB - Circulating immune complexes trigger type I interferons exacerbating disease in systemic lupus erythematosus. In this issue of Immunity, Henault et al. (2012) show that Fcγ-receptor, Toll-like receptor 9, and LC3 conspire to mold phagosomes into type I interferon signaling platforms.

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U2 - 10.1016/j.immuni.2012.11.009

DO - 10.1016/j.immuni.2012.11.009

M3 - Short survey

AN - SCOPUS:84870871486

SN - 1074-7613

VL - 37

SP - 947

EP - 949

JO - Immunity

JF - Immunity

IS - 6

ER -

Designing a Type I Interferon Signaling Phagosome

Abstract

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