Abstract
We have accumulated a significant body of evidence that support the hypothesis that aPL antibody-mediated disruption of annexin A5 crystallization is a mechanism for RSPL in the APS. Further investigation of the functions of the annexins in placental physiology and disease are likely to be productive areas for future research.
| Original language | English |
|---|---|
| Pages (from-to) | 77-81 |
| Number of pages | 5 |
| Journal | Thrombosis Research |
| Volume | 115 |
| Issue number | SUPPL. |
| State | Published - Feb 2005 |
| Externally published | Yes |