Correlation of serum IGF-I and IGFBP-1 and -3 to cardiovascular risk indicators and early carotid atherosclerosis in healthy middle-aged men

S. Boquist, G. Ruotolo, C. Skoglund-Andersson, R. Tang, J. Björkegren, M. G. Bond, U. De Faire, K. Brismar, A. Hamsten

Research output: Contribution to journalArticlepeer-review

39 Scopus citations

Abstract

Objectives: IGF-I, IGFBP-1 and IGFBP-3 are putative mediators in cardiovascular disease. The present study examined (i) the correlations of circulating IGF-I, IGFBP-1 and IGFBP-3 to established cardiovascular risk factors and signs of early atherosclerosis as reflected by ultrasound measurement of common carotid intima-media thickness (IMT), and (ii) whether serum concentrations of these analytes are modulated during alimentary lipaemia. Design: Cross-sectional clinical study. Patients: A biobank and clinical database based on 96 healthy Caucasian men, aged 50 years, with an apolipoprotein (apo) E3/E3 genotype, who had originally undergone investigations of postprandial lipoprotein metabolism was used for the study. Measurements: Total IGF-I, IGFBP-1 and IGFBP-3 were determined in serum by radioimmunoassay (RIA). Free IGF-I was measured by a commercial two-site immunoradiometric assay (IRMA). Results: In multivariate analyses, fasting serum free IGF-I correlated inversely with IMT and accounted for 5% of the variation in multiple R 2. When fasting serum IGFBP-1 was entered in the models instead of IGF-I, IGFBP-1 correlated positively with IMT and accounted for 6% of the variation in IMT. IGFBP-3 and total IGF-I were unrelated to IMT. There were no associations between free IGF-I and cardiovascular risk factors, whereas IGFBP-1 behaved like a component of the insulin resistance syndrome. Serum free IGF-I increased and IGFBP-1 decreased postprandially. Conclusion: The data indicate that serum free IGF-I and IGFBP-1 are implicated in early atherosclerosis.

Original languageEnglish
Pages (from-to)51-58
Number of pages8
JournalClinical Endocrinology
Volume68
Issue number1
DOIs
StatePublished - Jan 2008
Externally publishedYes

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