Abstract
Evidence exists for the presence of a neuro-anatomy that can produce cardiac vasospasm. Recent experimental and clinical data suggest that this neuro-anatomic mechanism can be activated by behavioral events in the psycho-social surround of the patient with coronary artery disease. I hypothesize that this mechanism can produce myocardial ischemia and can explain the variability in onset of clinical angina.
| Original language | English |
|---|---|
| Pages (from-to) | 369-374 |
| Number of pages | 6 |
| Journal | Medical Hypotheses |
| Volume | 24 |
| Issue number | 4 |
| DOIs | |
| State | Published - Dec 1987 |
| Externally published | Yes |
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