@article{d2dae4e0b76d4a74bd914a085fdd52b3,
title = "Cancer immunity derailed: PGE2 misconducts cDC1s",
abstract = "Type 1 conventional dendritic cells (cDC1s) are critical for CD8+ T cell-mediated tumor control. In this issue of Immunity, Bayerl et al.1 expose a mechanism leading to cancer progression where prostaglandin E2 induces dysfunctional cDC1s, which cannot coordinate CD8+ T cell migration and expansion.",
author = "Matthew Brown and Tran, {Michelle A.} and Nina Bhardwaj",
note = "Funding Information: M.B. is supported by the National Institute of Health (NIH) Public Health Service Institutional Research Training award AI07647. M.A.T. is supported by the NIH National Cancer Institute (NCI) F30 grant CA275269-01 as well as the Medical Scientist Training Program Training grant T32GM007280 from the NIH National Institute of General Medical Sciences. N.B. is an extramural member of the Parker Institute for Cancer Immunotherapy. N.B. is an extramural member of the Parker Institute for Cancer Immunotherapy (PICI), receives research funds from Regeneron, Antidote, and Dragonfly Therapeutics, and is on the advisory boards of Novartis, Rome Therapeutics, BreakBio, Carisma Therapeutics, Genotwin, the Cancer Research Institute, and on the Board of Directors for the American Association for Cancer Research (AACR). Funding Information: M.B. is supported by the National Institute of Health (NIH) Public Health Service Institutional Research Training award AI07647. M.A.T. is supported by the NIH National Cancer Institute (NCI) F30 grant CA275269-01 as well as the Medical Scientist Training Program Training grant T32GM007280 from the NIH National Institute of General Medical Sciences . N.B. is an extramural member of the Parker Institute for Cancer Immunotherapy. Publisher Copyright: {\textcopyright} 2023 Elsevier Inc.",
year = "2023",
month = jun,
day = "13",
doi = "10.1016/j.immuni.2023.05.015",
language = "English",
volume = "56",
pages = "1165--1167",
journal = "Immunity",
issn = "1074-7613",
publisher = "Cell Press",
number = "6",
}