Bortezomib disrupts tumour-dendritic cell interactions in myeloma and lymphoma: Therapeutic implications

Anjli Kukreja, Aisha Hutchinson, Amitabha Mazumder, David Vesole, Revathi Angitapalli, Sundar Jagannath, Owen A. O'Connor, Madhav V. Dhodapkar

Research output: Contribution to journalArticlepeer-review

32 Scopus citations

Abstract

Recent studies have shown that the interactions between tumour and dendritic cells (DCs) promote clonogenic growth of lymphoproliferative tumours, particularly myeloma. The present study showed that the proteasome inhibitor, bortezomib, disrupts this interaction. Targeting the drug to DCs was required for optimal suppression of tumour growth, including primary myeloma tumour progenitors in clonogenic assays. Bortezomib lead to dose-dependent induction of apoptosis in both myeloid and plasmacytoid DCs, and the sensitivity of DCs to bortezomib was comparable with that of tumour cells. These data suggest that disruption of tumour-DC interactions may contribute to the clinical effects of bortezomib.

Original languageEnglish
Pages (from-to)106-110
Number of pages5
JournalBritish Journal of Haematology
Volume136
Issue number1
DOIs
StatePublished - Jan 2007
Externally publishedYes

Keywords

  • Lymphoma
  • Myeloid dendritic cells
  • Myeloma
  • Plasmacytoid dendritic cells
  • Tumour microenvironment

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