BMAL1-Downregulation Aggravates Porphyromonas Gingivalis-Induced Atherosclerosis by Encouraging Oxidative Stress

Keyphrases

• Oxidative Stress 100%
• Bmal1 100%
• Porphyromonas Gingivalis (P. gingivalis) 100%
• Atherosclerotic Cardiovascular Disease (ASCVD) 71%
• NF-B 57%
• ApoE KO Mice 42%
• Inflammatory Response 28%
• Mouse Model 28%
• Atherosclerosis Progression 28%
• Circadian Clock 28%
• Metronidazole 28%
• Oxidative Stress Response 28%
• Melatonin 28%
• Arnt 28%
• Risk Factors 14%
• Causes of Mortality 14%
• Endothelial Cells 14%
• Novel Therapeutics 14%
• Knock-in Mouse Model 14%
• Effective Strategies 14%
• Mouse Brain 14%
• Cardiovascular System 14%
• Inflammation Response 14%
• Signaling Axis 14%
• Chronic Inflammation 14%
• Accelerated Atherosclerosis 14%
• Mouse Skeletal muscle 14%
• DNA Methyltransferase 1 (DNMT1) 14%
• Cre Mice 14%
• Pathogenic Bacteria 14%
• Human Aortic Endothelial Cells 14%
• Periodontitis 14%
• Clock Disruption 14%
• Joint Administration 14%

Immunology and Microbiology

• Atherosclerosis 100%
• Downregulation 100%
• Oxidative Stress 100%
• Porphyromonas gingivalis 100%
• Apolipoprotein E Knockout Mouse 42%
• Inflammation Response 28%
• Mouse Model 28%
• Circadian Rhythm 28%
• Muscle 28%
• Promoter Region 14%
• Knockout Mouse 14%
• Aortic Endothelial Cell 14%
• Periodontitis 14%
• Cardiovascular System 14%
• Infection 14%

Pharmacology, Toxicology and Pharmaceutical Science

• Atherosclerosis 100%
• Porphyromonas gingivalis 100%
• Coronary Artery Atherosclerosis 71%
• Apolipoprotein E Knockout Mouse 42%
• Inflammation 28%
• Mouse Model 28%
• Metronidazole 28%
• Synapsin I 28%
• Melatonin 28%
• Infection 14%
• Knockout Mouse 14%
• Chronic Inflammation 14%
• Periodontitis 14%