Autonomous Hyperprolactinemia in Tuberous Sclerosis

Zachary T. Bloomgarden, George W. Mclean, David Rabin

Research output: Contribution to journalArticlepeer-review

7 Scopus citations

Abstract

Amenorrhea and galactorrhea developed in a female patient with tuberous sclerosis. There was no evidence of a pituitary tumor; she had an abnormal EEG, and a computed tomographic scan showed multiple intracerebral calcifications but no lesions in the pituitary gland or hypothalamus. She had fixed hyperprolactinemia that was unresponsive to protirelin, chlorpromazine, levodopa, bromocriptine mesylate, or estrogen. The circulating prolactin may be of pituitary origin or may possibly be secreted ectopically by a hamartoma.

Original languageEnglish
Pages (from-to)1513-1515
Number of pages3
JournalArchives of Internal Medicine
Volume141
Issue number11
DOIs
StatePublished - Oct 1981
Externally publishedYes

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