Atlas on mechanisms in adverse reactions to food. The skin

H. A. Sampson

Atlas on mechanisms in adverse reactions to food. The skin

Research output: Contribution to journal › Short survey › peer-review

Abstract

Ingested food antigens rapidly cross the gastrointestinal barrier and reach pro-inflammatory cells in the skin. Food allergy provokes urticaria/angioedema by classical, Type I, IgE-mediated hypersensitivity. Food-induced atopic dermatitis is the result of non-classical, IgE-directed hypersensitivity involving resident mast cells, Langerhans cells, CD4⁺, TH₂ lymphocytes and monocytes. A form of gluten sensitivity provokes a characteristic eczematous-like rash and enteropathy (Dermatitis herpetiformis).

Original language	English
Pages (from-to)	47-51
Number of pages	5
Journal	Allergy: European Journal of Allergy and Clinical Immunology, Supplement
Volume	50
Issue number	20
State	Published - 1995
Externally published	Yes

Keywords

Angioedema
Atopic dermatitis
Dermatitis herpetiformis
Urticaria

Cite this

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AB - Ingested food antigens rapidly cross the gastrointestinal barrier and reach pro-inflammatory cells in the skin. Food allergy provokes urticaria/angioedema by classical, Type I, IgE-mediated hypersensitivity. Food-induced atopic dermatitis is the result of non-classical, IgE-directed hypersensitivity involving resident mast cells, Langerhans cells, CD4+, TH2 lymphocytes and monocytes. A form of gluten sensitivity provokes a characteristic eczematous-like rash and enteropathy (Dermatitis herpetiformis).

KW - Angioedema

KW - Atopic dermatitis

KW - Dermatitis herpetiformis

KW - Urticaria

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AN - SCOPUS:0028853265

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JO - Allergy: European Journal of Allergy and Clinical Immunology, Supplement

JF - Allergy: European Journal of Allergy and Clinical Immunology, Supplement

IS - 20

ER -

Atlas on mechanisms in adverse reactions to food. The skin

Abstract

Keywords

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