Abstract

Alopecia areata (AA) has been recently shown to also include T-helper cell type 2/IL-23 activation, in addition to T-helper cell type 1/IFN-skewing. The success of Jak inhibition together with IL-4Rα antagonism and limited response to IL-17A and PDE4 (protein) inhibition in AA are increasing our understanding of the complex immune interplay in AA. Trials testing targeted therapeutics are needed to further elucidate the pathogenic contribution of various cytokines.

Original languageEnglish
Pages (from-to)S55-S57
JournalJournal of Investigative Dermatology Symposium Proceedings
Volume20
Issue number1
DOIs
StatePublished - Nov 2020

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