Abstract
In this issue of Blood, Aǧar and colleagues present data for a novel explanation of how an antigenic target on β2GPI, a central protein in the APS disease process, can become available for binding by antibodies.
| Original language | English |
|---|---|
| Pages (from-to) | 1193-1194 |
| Number of pages | 2 |
| Journal | Blood |
| Volume | 116 |
| Issue number | 8 |
| DOIs |
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| State | Published - 26 Aug 2010 |
| Externally published | Yes |