A short corticosteroid course reduces symptoms and immunological alterations underlying long-COVID

  • Alberto Utrero-Rico
  • , María Ruiz-Ruigómez
  • , Rocío Laguna-Goya
  • , Estíbaliz Arrieta-Ortubay
  • , Marta Chivite-Lacaba
  • , Cecilia González-Cuadrado
  • , Antonio Lalueza
  • , Patricia Almendro-Vazquez
  • , Antonio Serrano
  • , José María Aguado
  • , Carlos Lumbreras
  • , Estela Paz-Artal

Research output: Contribution to journalArticlepeer-review

29 Scopus citations

Abstract

Despite the growing number of patients with persistent symptoms after acute SARS-CoV-2 infection, the pathophysiology underlying long-COVID is not yet well characterized, and there is no established therapy. We performed a deep immune profiling in nine patients with persistent symptoms (PSP), before and after a 4-day prednisone course, and five post-COVID-19 patients without persistent symptoms (NSP). PSP showed a perturbed distribution of circulating mononuclear cell populations. Symptoms in PSP were accompanied by a pro-inflammatory phenotype characterized by increased conventional dendritic cells and augmented expression of antigen presentation, co-stimulation, migration, and activation markers in monocytes. The adaptive immunity compartment in PSP showed a Th1-predominance, decreased naïve and regulatory T cells, and augmentation of the PD-1 exhaustion marker. These immune alterations reverted after the corticosteroid treatment and were maintained during the 4-month follow-up, and their normalization correlated with clinical amelioration. The current work highlights an immunopathogenic basis together with a possible role for steroids in the treatment for long-COVID.

Original languageEnglish
Article number1540
JournalBiomedicines
Volume9
Issue number11
DOIs
StatePublished - Nov 2021
Externally publishedYes

Keywords

  • Corticosteroids
  • Immunological alterations
  • Long-COVID

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