5-Aza-2′-deoxycytidine (decitabine) can relieve p21WAF1 repression in human acute myeloid leukemia by a mechanism involving release of histone deacetylase 1 (HDAC1) without requiring p21WAF1 promoter demethylation

Stuart A. Scott, Wei Feng Dong, Ryo Ichinohasama, Calley Hirsch, David Sheridan, Stephen E. Sanche, C. Ronald Geyer, John F. Decoteau

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Biochemistry, Genetics and Molecular Biology

Pharmacology, Toxicology and Pharmaceutical Science