Project Details

Description

Critically ill patients develop what is commonly referred to as 'stress diabetes' which is characterized by hyperglycemia and insulin resistance. Intensive insulin treatment (IIT) corrects hyperglycemia, decreases mortality and prevents the incidence of infection and sepsis in critically ill patients. Here we propose that the anti-inflammatory effects of insulin are exerted in part via the brain and would like to provide a rational for modifications to IIT and thus improve the care of critically ill patients.
StatusFinished
Effective start/end date1/01/1231/12/16

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