Adele Mossa

h-index

Research activity per year

Headline

POSTDOCTORAL FELLOW

Fingerprint

Dive into the research topics where Adele Mossa is active. These topic labels come from the works of this person. Together they form a unique fingerprint.

1 Similar Profiles

Interactome Analysis of the CC2D1A Scaffold Reveals Novel Neuronal Interactions and a Postsynaptic Role
Heller, A. T., Bhattacharya, A., Li, H., Turkalj, L., Thiyagarajan, S., Suzuki, E., Mossa, A., Zheng, H., Hao, L. & Manzini, M. C., 1 Apr 2026, In: Molecular and Cellular Proteomics. 25, 4, p. 101546 1 p.
Research output: Contribution to journal › Article › peer-review

Open Access
DEAD/DEAH-box RNA helicases shape the risk of neurodevelopmental disorders
Fiorenzani, C., Mossa, A. & De Rubeis, S., May 2025, In: Trends in Genetics. 41, 5, p. 437-449 13 p.
Research output: Contribution to journal › Review article › peer-review

Open Access
4 Scopus citations
Sex-specific perturbations of neuronal development caused by mutations in the autism risk gene DDX3X
Mossa, A., Dierdorff, L., Lukin, J., Garcia-Forn, M., Wang, W., Mamashli, F., Park, Y., Fiorenzani, C., Akpinar, Z., Kamps, J., Tatzelt, J., Wu, Z. & De Rubeis, S., Dec 2025, In: Nature Communications. 16, 1, 4512.
Research output: Contribution to journal › Article › peer-review

Open Access
7 Scopus citations
Developmental impaired Akt signaling in the Shank1 and Shank3 double knock-out mice
Mossa, A., Pagano, J., Ponzoni, L., Tozzi, A., Vezzoli, E., Sciaccaluga, M., Costa, C., Beretta, S., Francolini, M., Sala, M., Calabresi, P., Boeckers, T. M., Sala, C. & Verpelli, C., Jun 2021, In: Molecular Psychiatry. 26, 6, p. 1928-1944 17 p.
Research output: Contribution to journal › Article › peer-review

Open Access
31 Scopus citations
Molecular causes of sex-specific deficits in rodent models of neurodevelopmental disorders
Mossa, A. & Manzini, M. C., Jan 2021, In: Journal of Neuroscience Research. 99, 1, p. 37-56 20 p.
Research output: Contribution to journal › Review article › peer-review

Open Access
29 Scopus citations

Reports Summarize Life Science Findings from Rutgers University Robert Wood Johnson Medical School (Interactome Analysis of the CC2D1A Scaffold Reveals Novel Neuronal Interactions and a Postsynaptic Role)
Mossa, A.
25/09/25
1 item of Media coverage
Press/Media
Icahn School of Medicine at Mount Sinai Researchers Have Provided New Data on Autism (Sex-specific perturbations of neuronal development caused by mutations in the autism risk gene DDX3X)
Garcia-Forn, M., Wu, Z., De Rubeis, S. & Mossa, A.
4/06/25
1 item of Media coverage
Press/Media

Adele Mossa

Personal profile

Headline

Fingerprint

Collaborations and top research areas from the last five years

Dive into details

Interactome Analysis of the CC2D1A Scaffold Reveals Novel Neuronal Interactions and a Postsynaptic Role

DEAD/DEAH-box RNA helicases shape the risk of neurodevelopmental disorders

Sex-specific perturbations of neuronal development caused by mutations in the autism risk gene DDX3X

Developmental impaired Akt signaling in the Shank1 and Shank3 double knock-out mice

Molecular causes of sex-specific deficits in rodent models of neurodevelopmental disorders

Reports Summarize Life Science Findings from Rutgers University Robert Wood Johnson Medical School (Interactome Analysis of the CC2D1A Scaffold Reveals Novel Neuronal Interactions and a Postsynaptic Role)

Icahn School of Medicine at Mount Sinai Researchers Have Provided New Data on Autism (Sex-specific perturbations of neuronal development caused by mutations in the autism risk gene DDX3X)

Adele Mossa

Personal profile

Headline

Fingerprint

Collaborations and top research areas from the last five years

Research output

Interactome Analysis of the CC2D1A Scaffold Reveals Novel Neuronal Interactions and a Postsynaptic Role

DEAD/DEAH-box RNA helicases shape the risk of neurodevelopmental disorders

Sex-specific perturbations of neuronal development caused by mutations in the autism risk gene DDX3X

Developmental impaired Akt signaling in the Shank1 and Shank3 double knock-out mice

Molecular causes of sex-specific deficits in rodent models of neurodevelopmental disorders

Press/Media

Reports Summarize Life Science Findings from Rutgers University Robert Wood Johnson Medical School (Interactome Analysis of the CC2D1A Scaffold Reveals Novel Neuronal Interactions and a Postsynaptic Role)

Icahn School of Medicine at Mount Sinai Researchers Have Provided New Data on Autism (Sex-specific perturbations of neuronal development caused by mutations in the autism risk gene DDX3X)